Reprogramming - Globe Thesis

Keyword [Reprogramming] Result: 161 - 180 \| Page: 9 of 10
161.	The Mechanism Study Of Spermatogonial Stem Cell Reprogramming And De Novo Establishment Of Imprinting
162.	Generation Of Megakaryocytes By Chemical Compounds-Induced Lineage Reprogramming And The Underlying Molecular Mechanisms
163.	The Mechanism Of Pak4 In Maintaining The Pluripotency Of Mouse Embryonic Stem Cells
164.	How Chromatin State Affects The Influence Of Reprogramming Factors On Cell Fate
165.	Mechanisms Of CTCF And BRD4 On Gene Transcriptional Regulation And Somatic Cell Reprogramming
166.	The ARF2-PIF5 Functional Module Executes Transcriptional Reprogramming In ABS3-Mediated Senescence Pathway In Arabidopsis
167.	LncRNA Platr10 Regulates Stem Cell Pluripotency At The Translation Level Through The EIF4G2-MAP3K3 Pathway
168.	Development Of Three-dimensional Genome Software HiCHap And Its Application In The Study On Mouse Nuclear Transfer Reprogramming
169.	Reprogramming And Functions Of H2AK119ub1 And H3K27me3 In Mouse Gametes And Early Embryogenesis
170.	Comparative Study Of Reprogramming Efficiency And Pluripotency Of Horse,Donkey And Mule Induced Pluripotent Stem Cells
171.	Metabolic Reprogramming And Regulation Of Mouse Early Embryo Development
172.	Research On The DNA Methylation Regulation Mechanism On Self-renewal Of Induced Hepatic Stem Cells
173.	Newcastle Disease Virus-induced Nucleotide Metabolism Pathway Reprogramming In Infected Cells
174.	Study Of The Establishment Of Feeder-free Human Extended Pluripotent Stem Cells And Their Metabolic And Epigenetic Characteristics
175.	The Role Of Base Excision Repair In Cellular Reprogramming
176.	The Role And Underlying Mechanism Of Surfeit Locus Protein 4(Surf4) In Somatic Cell Reprogramming
177.	Nucleosome Reconfiguration During The Development Of Mouse Somatic Cell Nuclear Transfer Embryos
178.	Fast Chemical Reprogramming To Pluripotency
179.	Integrated Multiomic Characterization Of Fast Chemical Reprogramming
180.	Mitochondrial C1qbp Promotes Differentiation Of Effector CD8~+ T Cells Via Metabolic-epigenetic Reprogramming
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