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Keyword [CO poisoning]
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1. Series Studies Of The Biological Effects Of Hydrogen Gas
2. Hydrogen-rich Saline Reduces Delayed Neurologic Sequelae In Experimental Carbon Monoxide Toxicity
3. Clinical Studies On The Relation Of Creatine Kinase And Gravity In Acute CO Poisoning Case
4. MR Diffusion Weighted Imaging To Acute Carbon Monoxide Poisoning Of Rabbit Brain
5. Study On Expressions Of VCAM-1, IFN-γ And CD4~+ T Lymphocyte In Rat Models Of Delayed Neuropathological Sequelae After CO Poisoning And Developments After HBO Treatment
6. MR Perfusion Weighted Imaging Research In Acute Carbon Monoxide Poisoning Of Rabbit Brain
7. Study On Immune Factors And Neuroglia Cells In Rat Models Of Delayed Neuropathological Sequelae After CO Poisoning
8. Study On Demyelination And Immune Factors In Rat Models After CO Poisoning
9. The Clinical Analysis Of Delayed Encephalopathy After Carbon Monoxide Poisoning
10. HIF-1α And EPO Expression In Rat Brain’s Hippocampus Of The Delayed Effects Of Encephalopathy After Acute Carbon Monoxide Poisoning
11. The Expression Of Hypoxia-inducible Factor-1 Alpha And Heme Oxygenase-1 In Hippocampus Of Rats With Delayed Encephalopathy After Acute Carbon Monoxide Poisoning
12. Study Of The Changing Levels Of AP-1 And HO-1 In Hippocampus With Delayed Encephalopathy In Rats After Acute CO Poisoning
13. Expression Of Nrf2 And NQO1 In Hippocampus Of Rats With Delayed Encephalopathy Induced By Acute CO Poisoning And The Intervention Of Tert-butyl Hydroquinone On It
14. Molecular Mechanism Of Brain Damage Induced By Carbon Monoxide Poisoning And Feasibility Analysis Of Xingzhi Yinao Particles
15. Effect Of Nuclear Factor-?B Inhibitor PDTC On The Dynamic Changes Of INOS In Rats With Acute Carbon Monoxide Poisoning
16. Applcation Study Of The Diffusion Tensor Imaging In Patients With Dlayed Encephalopathy After Acute Carbon Monoxide Poisoning
17. Efficacy And Mechanism Of Fasudil Hydrochloride Combined With Hyperbaric Oxygen In The Treatment Of Brain Injury Caused By Carbon Monoxide Poisoning
18. CO-induced Brain Damage And The Role Of The TRPM2 Channel
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