| We studied the mechanism of resistance and the inheritance of resistance to Fusarium moniliform ear rot in maize using a susceptible maize genotype (inbred line 478), a resistant genotype (inbreds line R15) and the F1, F2, BC1, BC2 generations derived from the resistant inbred × susceptible inbred. The main results are as followed:1. Genes controlling maize ear rot resistance to Fusarium moniliform were at least 5 pairs by Estimating Least Gene Number method by Yu-Hua Ma.2. The results of 6 generation mean analysis indicated that dominance X dominance-epistatic effect wasn't dominant, and addictive effect, dominance effect and additive X dominance-epistatic effect were all super dominant in resistance to Fusarium moniliform maize ear rot. The value of addictive effect was larger than that of dominance effect, and the value of additive X dominance-epistatic effect was larger than that of addictive and dominance effects. So we think that addictive effect and additive X dominance-epistatic effect were dominant in the genetic effect of resistance to Fusariummoniliform maize ear rot.3. The result of major and minor gene genetic model analysis showed the resistance of maize ear rot was controlled by 2 pairs additive major genes and polygenes, and the values of positive effect were larger than negative ones in the 2 pairs additive major genes, and the values of addictive polygene positive effect were also larger than the ones of dominance polygene negative effect in polygene addictive-dominance effect model, which was almost similar to the result of generation mean analysis.4. The results also revealed that agricultural characters investigated such as spike length, spike width, spike rows and kernel depth, etc. had less correlation to resistance to Fusarium moniliform maize ear rot, which showed that resistance to maize ear rot was mainly controlled by genetic factors, and phenotype characters can't be a select index in breeding the species resistant to Fusarium moniliform maize ear rot.
|
PDF Full Text Request