Relationship Acute Myocardium Infract,Diseased Vessel And Infraction Site Relative To ECG ?,? And AVF Change

Background:Acute myocardial infarction characterized the symptom of the chest discomfort duration more than 30 minutes that radiate to throat,jaw,left shoulder,and left arm.The inducing factors are including: labour exertion,stress,and heavy meals.MI suffering patient is not getting better with the rest or sublingual nitro-glycerine.ECG test shows ST elevation in the leads which corresponding the site of infarction.Coronary angiography is the golden standard to assess the degree of artery stenosis.The biomarkers of the myocardial infarction such CK-MB are elevated in the setting of acute MI.complication of the acute myocardial infarction such arrhythmia,rapture of the wall of the heart and inferior MI hypotension.Objective of the study:The goal of the study is to find the relationship between culprit arteries,site of myocardial infarct and ECG changes in the inferior leads of the 12 leads of EKG.Methods:Clinical records of the patient diagnosis with acute inferior ST segment myocardial infarction those admissions in the department of cardiovascular medicine of the Jilin university from 2017 January to 2017 December was studied retrospectively.Ethical approval was obtain from the research ethics committee of the first hospital of Jilin university.Data will be analyzed using SPSS version 18.RESULTS:We enrolled 61 patients in our study with diagnosis of acute inferior of myocardial infarction from 2017 January to 2017 December.When compared the incidence of ST segment elevation in lead ?,? and aVF between the three diseased artery(RCA,LAD & LAD/RCA),there was a significant difference(P = 0.025)indicating that 33/61(54.1%)patients who had ST-Elevation were RCAConclusion: In conclusion,ST segment elevation in leads ?,? and aVF in acute inferior wall of myocardial infarction.The right coronary artery occlusion was at large stenosis artery in acute inferior myocardial infarction in leads ?,? and aVF with ST segment elevation in ?>? sugest left circumflex artery occlusion.