| In recent years, disease-resistance is the root case in the increase of output of maize, which is one of the most important cereal crops and forage crops worldwide. Thus researches on disease-resistance plants are turning into the hot issues in genetics and pathology. As an important disease of maize, ear rot in maize occurs in maize planting areas all over the world, becoming one of the main diseases of maize. The plentiful fungous toxins produced by Fusarium moniliforme, exert serious and poisonous side-effects on human beings and poultry. Which are demonstrated by a series of disease occurring to them after taking food with such toxins? Fusarium moniliforme there upon is regarded as the prominent problem of maize production in our country which calls for imminent way of resolving. Scholars at home and abroad divide the resistance of cobrot of maize into silk resistance and the kernel resistance, and lots of researches have been carried out on them. Seen that cob of maize is a channel through which germs infect, as well as a main passageway through which rob enter into ear, we deem that the resistance of rob is also prominent in the point of ear rot of maize. With the actual production of maize as the point of departure, the group of reaches topic carried out reaches. On genetic resistance of cob rot in maize engendered by Fusarium moniliforme (including kernel and cob rot caused by Fusarium moniliforme), based on the separation and purification of the advantage fungus in Henan region and the selection of antigens. Through continuous years of inoculation and SSR marker analysis in different places and different backgrounds the following conclusions have been mainly achieved:1. Cob resistanceThe inheritance of resistance to cob rot caused by Fusarium moniliforme was studied on progeny derived from the crosses between resistant inbred (BT-1) and susceptible inbred (xiongzhang and xi502) by improved kernel-stab inoculation method from 2007 to 2008. Six generations ( the parents , F1,F2 and their backcrosses) derived from the cross of BT-1 and xiongzhang,xi502 were used to study the inheritance of resistance to Fusarium moniliforme cob rot. F2:3 families are produced by the self-bred of each indicidual plant of F2 groups produced by BT and xiongzhang, thus a new family are divided into four repetitions and sowed in different places in summer and spring, and the disease-resistance is the average of the disease-susceptibility of the four repetitions. Make use of BT-1 and bear the F2 groups, F2: 3 families to the major resistance QTL cob rot position in chromosomes2. 3, the chromosome 3 is the largest value of resistance cob rot in maize, which could explain 28.33 % of the phenotypic variation.There are same results of resistance cob rot in different years and different places. It is good for fine maping and map-based cloning.2. Kernel resistanceThe inheritance of resistance to cob rot caused by Fusarium moniliforme was studied in progeny derived from the crosses between resistant inbred (BT-1) and susceptible inbred (xiongzhang and xi502) by improved kernel-stab inoculation method in 2008. One of the major QTLs was located on chromosome 4, which could explain 24.51% of the phenotypic variation. At the same time, using resistant line BT-1 and susceptible line N6 construct RIL population. The phenotypic identification and resistant QTL analysis of ear rot in many years. Multiple repeats by RIL, is the same as the result of F2 groups by BT-1 and xi502 in kernel resistance.
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