The Dysfunction Of Immunifaction In Patients With Spontaneous Rupture Of Hepatocellular Carcinoma

Objective:The purpose of this study is to examine swallow function of macrophage; furthermore investigate the mechanism related to spontaneous rupture of hepatocellular carcinoma(HCC).Method:In this study,we used to look-forward experimentation of pairing analysis to collect 10 specimens from rupture HCC and 10 cases non-rapture HCC.Quantificationally investigate to expressions of hepatic marchohage(CD68) and its CD18and CD16 of surface receptors in immunohistochemcal analysis.Qualitatively analysis to locations and distributions of hepatic macrophage(CD68) and its CD18 and CD16 of surface receptors immunohistochemical analysis.Results:Quanitificationally investigate to expressions of hepatic macrophage(CD68) in immunohistochemical analysis from cases with ruptured HCC and non-ruptures HCC:â‘ Expressions of macrophage (CD68) and its CD18 and CD16 of surface receptors from carcinoma tissue is lesser cases with adjacent non-cancerous liver tissues in groups with non-ruptured HCC markedly;â‘¡Expression of macrophage(CD68) and its CD18 and CD16 of surface receptors from liver carcinoma tissues is lesser cases with adjacent non-cancerous live tissues in groups with adjacent non-cancerous liver tissues in groups with HCC markedly;â‘¢Expression of macrophage(CD68) and its CD18 and CD16 of surface receptors from groups with ruptured HCC is lesser cases with non-ruptured HCC is liver carcinoma tissues markedly.â‘£Expression of macrophage(CD68) from groups with ruptured HCC is lesser cases with non-raptured HCC in liver carcinoma tissues markedly.Conclusion:â‘ Expressions of macrophage(CD68) and its CD18 and CD16 of surface receptors in patients from spontaneous rupture of hepatocellular carcinoma is lesser cases with non-ruptured HCC.Its function of swallow was injured possible;â‘¡Expressions and distributions of macrophage(CD68) and its CD18 and CD16 of surface receptors in patients from liver carcinoma tissues is lesser cases with adjacent non-cancerous liver tissues,which result in low of tissues resistance.