| 1Research ObjectivesDetermination to explore the impact of different intensity intermittent running training onenergy metabolism in rats metabolizing enzymes and oxidation of matrix-related indicators onmyocardial energy metabolism, the development of exercise training intensity in order to providetheoretical support.2Experimental MethodsSelect the4-week-old male wistar rats70(Shandong Lukang Pharmaceutical Co.,Ltd.),average weight of67.99g±5.83g were randomly divided into seven groups (n=10), that is,quiet in the control group and exercise group, exercisegroup exercise training intensity group,65%and70%,75%,80%,85%and90%group. Treadmill Training for9weeks, training sixdays a week, training three times in each group, each of not less than10minutes, an interval of30minutes, the last24hours after exercise, and destroying the spinal cord law killed afterdissection, quickly removed out of the heart muscle, saline rinse clean, and set to-84°Crefrigerator under test. Preparation of tissue homogenates were measured in rat cardiac muscleglycogen content, succinate dehydrogenase (SDH), malate dehydrogenase (MDH), lactatedehydrogenase (LDH), Ca2+ATPase enzyme, lactic acid content. All data SPSS17.0software forstatistical analysis.3Results3.1Rat cardiac muscle glycogen content80%and85%of cardiac muscle glycogen content was significantly higher than that in thecontrol group (P <0.05);80%and85%of the cardiac muscle glycogen content was significantlyhigher than the65%group (P <0.05); controlgroup,65%,70%, between75%and90%in cardiacmuscle glycogen content was significantly difference.3.2rat cardiac SDH activity90%of myocardial SDH activity was significantly higher than that in the control groupand65%group (P<0.05); control group,65%,70%,75%, between80%and85%of myocardialactivity of SDH statistically significant.3.3rat heart MDH activity65%,70%,75%,80%,85%and90%myocardial MDH activity were significantly higher (P<0.05);85%of myocardial MDH activity, but the movement MDH activity in myocardium between the groups was no significant difference.3.4rat cardiac LDH activity65%,70%,75%,80%,85%and90%of myocardial LDH activity were significantly higher (P<0.05);90%of myocardial LDH activity was significantly higher than65%and70%(P <0.05);65%,70%,75%,80%and85%group myocardial LDH activity increased trend but with each other without significant differences.3.5rat myocardial Ca2+ATPase activityThe cardiac group,65%and70%,75%,80%and85%group of Ca2+ATPase activity was significantly higher (P<0.05);85%of myocardial Ca2+ATPase activity of the highest;90%of myocardial Ca2+ATPase enzyme is slightly higher, but no significant difference.3.6rat cardiac LD content90%of LD content was significantly higher (P<0.05); no significant differences between other groups.4Conclusion4.165%between V02max,70%between V02max and75%of VO2max, moderate-intensity treadmill training did not significantly increase the glycogen content of rat cardiac muscle, while85%VO2max intensity of long-term training can significantly increase rat cardiac muscle sugarthe original content.90%of V02max, long-term treadmill training is not conducive to the rat cardiac muscle glycogen recovery and reserves.4.2different intensity treadmill training can significantly increase rat myocardial aerobic metabolic enzyme SDH, the number and activity of MDH,85%VO2max, long-term treadmill training can improve the enzyme activity of rat myocardial aerobic metabolism, is conducive to myocardial the development of aerobic metabolic activity.4.3different intensity treadmill training can significantly improve the anaerobic metabolism of rat cardiac enzyme LDH activity,90%VO2max LDH activity.90%of VO2max, long-term treadmill training can make the rat heart muscle glycogen, anaerobic glycolysis for energy, resulting in LD, and24hours can not be fully restored.4.465%between V02max,70%between VO2max,75%between V02max,80%between V02max and85%VO2max long-term treadmill running can significantly increase the activity of rat heart of Ca2+ATPase,85%VO2max Ca2+ATPase activity of the highest.90%of VO2max long-term treadmill training resulted in a rat cardiac metabolic disorders, is not conducive to the recovery of the Ca2+ATPase. |