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Idiopathic Menorrhagia Endometrial Estrogen And Progesterone Receptor Analysis

Posted on:2011-07-23Degree:MasterType:Thesis
Country:ChinaCandidate:L J LiFull Text:PDF
GTID:2154360308482017Subject:Obstetrics and gynecology
Abstract/Summary:PDF Full Text Request
Objective: To investigate patients with idiopathic menorrhagia endometrial estrogen receptor and progesterone receptor expression, of idiopathic menorrhagia in patients with endometrial estrogen receptor and progesterone receptor expressionMethods: 2007-2009, out-patient diagnosis of idiopathic menorrhagia in women, 60 cases were detected by immunohistochemistry Elivision endometrial glands and stroma of estrogen receptor and progesterone receptor expression while comparing normal endometrium and simple hyperplasia of the endometrium. Experimental data with X±S expression. Statistical analysis of experimental data, statistical software applications SPSS10.0 t test, statistically significant levels were set at P <0.05. (With P <0.05 as statistically significant level)Results: In normal endometrial ER, PR periodical, hyperplasia > the secretory phase, reached a peak in the late proliferative phase. Idiopathic menorrhagia ER, PR the same periodical, but in the proliferative phase and secretory phase were higher than the normal endometrium. Simple hyperplasia of the endometrium ER, PR positive integral> idiopathic menorrhagia proliferative phase> normal endometrial proliferation. Conclusion: Because of idiopathic menorrhagia ER, PR in the proliferative and secretory phase were higher than the same period in normal endometrium, the cause of idiopathic menorrhagia excessive endometrial hyperplasia occurred Secretion shortage; as simple hyperplasia of the endometrium ER, PR positive integral>idiopathic menorrhagia proliferative phase>normal endometrial proliferation, it led to excessive proliferation of endometrial phenomenon.
Keywords/Search Tags:idiopathic menorrhagia, endometrium, estrogen and progesterone receptor, immunohistochemistry, simple hyperplasia
PDF Full Text Request
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