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Discussion On Insulin Resistance Of Type 2 Diabetes Mellitus From Spleen Deficiency And Phlegm

Posted on:2016-08-30Degree:MasterType:Thesis
Country:ChinaCandidate:X F MaoFull Text:PDF
GTID:2134330470973835Subject:Integrative Medicine
Abstract/Summary:PDF Full Text Request
Object: Through consulting,summarizing and thinking about the ancient and modern document on insulin resistance in type 2 diabetes mellitus and discussing the actual work in clinical problems, I think the opinion that treating the insulin resistance in type 2 Diabetes mellitus from spleen deficiency,phlegm and blood stasis is important,feasible and reasonable. Method: Basing on the theory of Traditional Chinese Medicine and the western medicine’s understanding about insulin resistance, according to the importantpathophysiological mechanism of insulin resistance is spleen deficiency, respectively discussing from the forming process that wet phlegm and blood stasis in patients with insulin resistance in type 2 diabetes mellitus, the important role that phlegm and blood stasis in insulin resistance about type 2 diabetes mellitus, strengthening spleen, preventing phlegm and blood quickening is the most important, reasonable and feasible treatment for type 2 diabetes about insulin resistance. Finally Combinning with the typical clinical case thattreating from the type 2 diabetes mellitus about insulin resistance form spleen deficiency,phlegm and blood stasis, making the arguments in the thesis obtain further analysed,discussed and confirmed. Conclusion:Spleen deficiency,phlegm and blood stasis plays a quite important role in the occurrence and development of type 2 diabetes mellitus about insulin resistance, treating the insulin resistance in type 2 diabetes mellitus from spleen deficiency,phlegm and blood stasis has its theoretical and clinical basis,regulating zang-fu qi-blood centre on spleen can improve clinical efficacy.
Keywords/Search Tags:spleen deficiency, phlegm and blood stasis, insulin resistance, type 2 diabetes mellitus
PDF Full Text Request
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